What Western glomerulonephritis can eat?

You should be open to the Hospital of Traditional Chinese Medicine with ~
should light, avoid spicy salty!
1 can be divided into common type of chronic nephritis, kidney disease type, type of hypertension and asymptomatic type. Mild discomfort was not significant, self-limiting or maintain a stable after years and years. Severe cases may have hematuria, hypertension, edema, proteinuria, anemia, and even a few months progress to renal failure, uremic stage. The general progress of the disease is slow, relatively stable. Dietary requirements should be based on the different stages of disease development and clinical manifestation of discrimination.
(1) asymptomatic, no hematuria or proteinuria less than 1 gram per day to 2 grams of the patient may be given general diet, a little salt restriction.
(2) cause more urinary protein plasma protein is low, without azotemia who may be appropriate to increase the protein supply to 1 gram per kg body weight per day subject to regular checks of renal function status, protein intake at any time to adjust intake.

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(3) impaired renal function should limit the amount of protein people, the daily control of 30 g to 40 g (including the staple food containing protein). Those with mild renal dysfunction according to the daily per kilogram of body weight o. 7 g ~ o. Supply 8 grams. In the limited range of high quality protein foods such as milk, eggs, lean meat, etc., the best days of total protein accounted for more than 50%.
(4) sodium intake depends on the degree of edema and hypertension, the general principle of food is appropriate to light.
(5) who should be cautious of high potassium high potassium vegetables, fruit.
(6) to be used hyperlipidemia less fat, low cholesterol diet.
(7) avoid using alcoholic beverages and irritating foods.
symptomatic treatment of diuretic can be swelling, lower blood pressure. Commonly used thiazide diuretics (eg, Hydrochlorothiazide 25mg, 2 or 3 times a day), only when necessary to loop diuretics such as furosemide (fursemide) 20 ~ 60mg/d, or sub-sub-oral injection. Lee calligraphy still not satisfied after the blood pressure when the modern system, can be added with calcium channel blockers such as nifedipine 20 ~ 40mg/d, split vasodilators, such as oral or hydrazine phthalazine (hydralazme) 25mg, 3 times a day . However, potassium diuretic drugs (such as triamterene and spironolactone) and the conversion of angiotensin converting enzyme inhibitors (such as the heart thiol Jiabing acid) should be used with caution in oliguria to E induced hyperkalemia. answer added The most important thing is according to doctor's requests. seeking answers is the best way to supplement treatment you do seek medical advice as a prescription is really better. right. so listen, may delay the disease.
the hospital general medicine is hormones. because the hormone and quick, but it belongs to the temporary control of the disease, not a direct treatment of kidney disease, so most patients will appear repeatedly, is A drug dependent. in order to eradicate the disease would be better to use traditional Chinese medicine, Chinese medicine with western medicine so it may not be obvious, but only until the Chinese medicine is fundamental. There are also non-drug treatment is also very good The.
glomerulonephritis is a common disease, mostly occurs in children, to hematuria, proteinuria, edema, hypertension as the main performance. Clinical and sub-acute glomerulonephritis and chronic glomerulonephritis. Short course of acute nephritis (mostly less than one year), through the active treatment, the prognosis is good. Long course of chronic nephritis, recurrent, prognosis is poor, if not actively treated, and finally the majority develop chronic renal failure or uremia. A Zu drugs,drugs question and answers

group B hemolytic streptococcus is the most important bacterial cause glomerulonephritis, a Zu hemolytic streptococcus group B infection, tonsillitis, scarlet fever after the bacterial antigens and human roundworm the body's antibody (immunoglobulin), forming antigen-antibody immune complex deposition in the glomerular basement membrane, can cause a series of inflammatory reactions. The acute stage, can be found in the blood of patients immune complexes, using immunofluorescence methods can be found in the glomerular basement membrane of irregular granular deposits (containing immunoglobulin G, complement 3, complement 4, prepared Solution elements, etc.). Electron microscopy can see the hump-like immune complexes were deposited on the basement membrane. Streptococcus

addition, other bacteria or viruses can also lead to glomerulonephritis, such as pneumococcus, staphylococcus, hepatitis virus, influenza virus and fungi, protozoa, etc., can cause kidney ball nephritis. The composition of bacteria or virus antigen and antibody to form immune complex deposition in glomeruli, inflammation can be formed. The symptoms and post-streptococcal glomerulonephritis caused similar but less serious symptoms.

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