Will the emphysema patients, have asthma, has been coughing,

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Oxygen therapy in chronic bronchitis, emphysema, pulmonary heart disease is part of the basic pathology of small airway obstruction, to a certain extent that is caused by hypoxia, often associated with carbon dioxide retention can cause respiratory failure type Ⅱ . Ⅰ respiratory failure due to hypoxia, chronic bronchitis, emphysema can occur early type Ⅰ respiratory failure, pulmonary heart disease, but in particular after the late rare. Airway obstruction resulting from the lack of pulmonary ventilation, and then there hypoxemia, arterial partial pressure of oxygen to (PaO2), arterial oxygen saturation (SaO2) and arterial oxygen content (CaO2) down, and then causes tissue hypoxia. Under normal circumstances, from the atmosphere to the pressure, lung, blood, tissue oxygen drop between the tonsure, the mitochondrial levels of the need for 6. 67kpa (50mmhg) the partial pressure of oxygen to maintain normal metabolism, in general, the more serious lower oxygen hyperlipidemia, PaO2 <6.67kpa (50mmgh) can lead to tissue hypoxia. In addition chronic bronchitis, emphysema, pulmonary heart disease patients often had cardiac insufficiency, blood components and acid-base status change, influence blood oxygen transport, and the combination of oxygen with hemoglobin and release, can eventually lead to tissue hypoxia, which affect the body normal physiological functions or life-threatening, so chronic bronchitis, emphysema, pulmonary heart disease in a timely manner the right oxygen therapy, in fact, an important therapeutic measures. drugs,drugs question and answers

expectorant and aerosol inhalation

chronic bronchitis, emphysema, bronchial mucus gland hyperplasia, hypertrophy, secretory hyperactivity, sputum more. Exudation increased after infection, further increased sputum, often more viscous, difficult or unable to cough and increased airway obstruction and infection. It phlegm is an important aspect of treatment, oral expectorants are the most common method than inhalation expectorant, the Shang Youliang good airway humidification, administration and anti-inflammatory effect.

bronchodilators

treatment of chronic bronchitis, emphysema, pulmonary heart disease, due to a variety of adverse external factors, incentives, and the disorder of physiological processes, bronchial there is inflammation and spasm. Thus patients with a sense of wheezing, difficulty breathing, cyanosis appeared, and the lungs could be heard at this time how much experience the range of wet and dry rales, wheeze. Pulmonary function tests decreased lung capacity can be found in practice shows that there is increased airway resistance, cause obstruction, plays for the subsequent lack of oxygen, carbon dioxide retention of the direct cause of the spasm is caused by pulmonary artery, pulmonary hypertension, right ventricle and pulmonary heart disease basis. In order to alleviate clinical symptoms, to correct airway spasm, blocking subsequent blood gas and secondary cardiovascular disease, timely and correct use of bronchodilator is an important aspect of treatment.
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bronchodilator variety, developed quickly, with some side effects and efficacy are not clear of drugs, such as ephedrine, isoproterenol gradually less so. Clinical use in recent years, more alkaline for the tea category, anticholinergic, β2 agonist class, adrenal cortex hormones, results are good. If properly grasp, some side effects not.



vasodilator therapy of chronic bronchitis, emphysema, pulmonary heart disease, due to long-term hypoxia, leading to contraction of small pulmonary arteries, the resistance increased. Acute attack of contraction increased, resulting in pulmonary hypertension, right heart failure. Can at this time cardiac diuretic therapy, but the heart of the cardiac oxygen sensitive, prone to cause poisoning, diuretics can cause electrolyte imbalance, it is subject to some restrictions apply. May also give clinical vasodilator therapy, often receive better effect.

vasodilator of pulmonary arteries can be expanded to reduce pulmonary artery pressure, pulmonary vascular resistance to decrease. Peripheral vein can also expand the capacity increase. Thus reduce the load before and after the heart and reduce oxygen consumption. Phentolamine and nifedipine also the role of bronchial relaxation, can reduce airway resistance, improving ventilation, increased blood pressure, lower arterial carbon dioxide partial pressure, lower arterial carbon dioxide partial pressure, so that condition has become better. drugs,drugs question and answers



respiratory stimulant treatment of chronic bronchitis, emphysema, cardiopulmonary disease, respiratory failure may occur, resulting in severe hypoxia and carbon dioxide retention can occur, including the nervous system awareness of symptoms, at this time, inhibition of the respiratory center is also known as pulmonary encephalopathy, the prognosis is often poor. The essence of the treatment to improve the ventilation, increasing oxygen inhalation and carbon dioxide emissions, so you can use the respiratory stimulant treatment to excitement or peripheral chemoreceptor respiratory center to increase the respiratory drive to increase tidal volume and respiratory rate, ventilation increased to alleviate the oxygen and carbon dioxide retention purposes. These patients have paralysis of respiratory center, but also increase airway resistance, after the use of respiratory stimulants although the increase in ventilation, but the respiratory muscles for extra power too great to overcome airway resistance was significantly increased oxygen consumption, hypoxia and Carbon dioxide retention may not be alleviated. Therefore, before the need to first give bronchodilator drug therapy to minimize airway resistance. Be observed after treatment in patients with consciousness, breathing and cyanosis, eyelash reflex, arterial blood gas analysis and other indexes, improvement can continue medication, such as invalid can be disabled. The role of respiratory stimulants in patients with a clear advantage at this time may encourage patients to cough, to promote expectoration and maintain airway, breathing muscles for power saving.

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